Q. I noticed that GUTPro™ contains L. plantarum that is not D-lactate free. Why?
A. GUTPro™ contains L. plantarum for a very good reason. It has actually been proven to reverse acidosis. L. plantarum initially produces L(+)-lactate, and then switches to producing D(-)-lactate, allowing for equilibrium to be reached. The production of D(-)-lactate in L. plantarum is linked to the biosynthesis of the cell wall.
Q. What about L. salavarius? Is this strain D-lactate free?
A. L. salavarius primarily produces L-lactate and this is why it commonly referred to as “d-lactate free”. But, in reality it does produce a small amount of D-lactate. No studies have shown that this strain has ever caused acidosis. The case is similar with L. gasseri, which predominantly makes L(+)-lactate during the growth phase and switches to D(-)-lactate when the growth cycle plateaus.
Q. I heard that probiotics that contain D-lactate producing strains cause acidosis.
A. Just because a strain produces D-lactate does not mean that it will contribute to acidosis. GUTPro™ is formulated with L. plantarum which produces both D-lactate and L-lactate. L. plantarum has actually been proven to reverse acidosis.
Q. Isn’t it true that some probiotics can cause D-lactic acidosis?
A. Yes. Several studies have shown that L. acidophilus has caused acidosis in individuals with compromised digestive systems. That is why GUTPro™ was specifically designed without this popular strain of probiotic.
Q. When does D-lactic acidosis occur?
A. D-lactic acidosis occurs when the body is unable to properly metabolize excess D(-)-lactate.
Q. What are the symptoms of D-lactic acidosis?
A. Impaired mental status is a universal feature in D-lactic acidosis. Some of the common neurological symptoms include:
- Impaired motor coordination
- Slurred speech
- Aggressive or hostile behavior, agitation
- Stupor, ataxia and gait disturbance
- Nystagmus (involuntary eye movement)
- Inability to concentrate
- Carbohydrate craving
- Unhappiness and irritability
- Headache, bruxism and opisthotonus
- Hyperventilation and tachypnoea
- Nausea or pallor
Q. What else can contribute to D-lactic acidosis?
- Carbohydrate malabsorption
- Ingestion of large amounts of carbohydrates
- Reduced gut motility
- Impaired D(-)-lactate metabolism
Q. Can a urine test diagnosis D-lactic acidosis?
A. Elevated urine D(-)-lactate levels are often confused with D(-)-lactic acidosis. Elevated urine D(-)-lactate levels may be due to a gastrointestinal microbiota predominated by species that produce high amounts of D(-)-lactate.
Q. Can diet help with D-lactic acidosis?
A. The biggest factor in successfully treating D-lactic acidosis is following the proper diet, such as the GAPS Diet (which was derived from the Specific Carbohydrate Diet, SCD). Simple sugars such as glucose, fructose, and lactose have been shown to promote excessive D(-)-lactate generation by intestinal bacteria and in some cases to precipitate acidotic symptoms.
Q. Where can I find out more details about GUTPro™ custom probiotic?
A. Visit the following webpage to learn more about GUTPro™ custom probiotic: